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However in our opinion, the low probability of MS coincidence with another rare disease, together with the animal studies where MS is never a consideration, makes it more likely that these human examples align with an inside-out model (Figure 2), where a primary biochemical derangement of myelin, coupled with an immune predisposition, culminated in a MS-like phenotype

This leads to mitochondrial DNA damage and endothelial injury [167]

BAD-dependent regulation of fuel metabolism and K(ATP) channel activity confers resistance to epileptic seizures

10.3390/nu10091132 160

A unifying mechanism of ketogenic diet action: the multiple roles of nicotinamide adenine dinucleotide