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The genesis of acetic acid-induced gastric lesions is a multifactorial process which starts mainly with the depletion of gastric wall mucous content [41]

The main anthocyanins were significantly down-regulated in the heat sensitive cultivars under high temperature conditions

Clinical trials of glutaminase inhibitors ( Tables 2 ), particularly telaglenastat (CB-839), have shown that targeting cancer metabolism via GLS inhibition is feasible and generally well tolerated, with consistent evidence of on-target activity