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The interactions between citrulline and molecules associated with the JAK/STAT axis, together with the regulatory effects of urea cycle-related enzymes on the activation of inflammatory macrophages, suggest that the iNOS/NO axis may exert feedback regulation on pro-inflammatory signaling in different contexts ( At the intervention level, regulating the metabolic partitioning of Arg can alter macrophage phenotypes and inflammatory intensity
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Dec 1983;141 Suppl:453-463
Increased HSP90 expression has been observed in MASLD, where it contributes to lipid imbalance and metabolic dysfunction
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