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By neutralizing free radicals, it helps to mitigate cellular damage that can result in chronic inflammation

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Moreover, microglia return to their resting morphology after systemic bacterial infection, even though the brain shows elevated proinflammatory mediators, including INF-, IL-1, and IL-12 (Puntener et al., 2012), suggesting microglial inflammatory responses are not fully resolved and remain in a primed state that can augment microglial responses in the context of subsequent stimuli