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A single molecular entity simultaneously activates both the GLP-1 receptor and the amylin receptor (calcitonin receptor complex), combining two complementary satiety and metabolic pathways: GLP-1 receptor activation : Glucose-dependent insulin secretion, glucagon suppression, delayed gastric emptying, and central appetite reduction through hypothalamic signaling Amylin receptor activation : Additional satiety signaling through the area postrema, complementary gastric emptying delay, and glucagon suppression through a distinct mechanism The unimolecular design means both receptor activations occur at a fixed ratio determined by the molecular structure

The main character of AD is irreversible brain dysfunction, manifested as deterioration of cognitive functions, motor deficits, behavioral disturbances etc

And then, uh Evan, quickly, uh I want to get you out on a question that I'm going to guess you are asked by investors, which is okay, talk about GLP-1s, this is all we talk about

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