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But they work downstream of the CXXC5 blockade

Using this model, we now demonstrate that Trkb deletion-induced iSPN dysfunction leads to a hyperdopaminergic state in the pre-symptomatic phase, before motor dysfunction onset, reminiscent of the dopaminergic dysfunction observed in the brains of patients with HD 3,4,5

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TB-500 is studied primarily for cell migration through actin regulation the synthetic Thymosin Beta-4 fragment's mechanism involves binding G-actin and influencing the cytoskeletal dynamics that drive cells to physically move toward sites of injury

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