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therefore, assessing the methodological quality or risk of bias of the included studies was not mandatory

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Studies on mechanisms of anti-atherogenic activity of HDL, mainly reverse cholesterol transport, have also led to the discovery of dysfunctional HDL, developed in inflammatory and/or oxidative stress conditions (as well as a result of glycation and other processes), with limited or nullified anti-atherosclerotic activity, or even with pro-atherosclerotic properties [41]

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