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Therapeutically, p53 activation dualistically regulates apoptosis and ferroptosis to suppress PCa growth, but ferroptosis-driven macrophage polarization in the TME may inadvertently promote tumor metastasis [Figure 2A], emphasizing the need for context-specific modulation to balance efficacy and paradoxical outcomes

[DOI] [PMC free article] [PubMed] [Google Scholar] 85.Ghosh B., Zhang C., Smith G

Mitochondrial division inhibitor 1 (mdivi-1) protects neurons against excitotoxicity through the modulation of mitochondrial function and Intracellular Ca 2+ signaling

O., Osaki, K

Understanding these differences helps consumers evaluate whether patch-based supplements align with their individual needs and preferences