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doi: 10.7717/peerj.8893 140 GuoLXiaoPZhangXYangYYangMWangTet al

The data supports this

These findings support the hypothesis that weight loss is largely exposure-driven and may be comparable across agents at specific dose levels

It also engages other intracellular pathways, including -arrestin-mediated MAPK signaling, where -arrestin desensitizes GPCRs by blocking G protein coupling and acts as a scaffold to activate mitogen-activated protein kinases (like ERK), promoting longer-term effects such as gene expression and cell survival by regulating gene transcription, independent of G protein signaling [22]

Secondly, GLP-1 receptor agonists carry important safety considerations including risk of pancreatitis, gallbladder disease, and potential thyroid C-cell tumours (based on rodent studies, though human relevance remains uncertain)