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Geresnis atsistatymas siejamas su maesniu oksidaciniu stresu ir efektyvesne lsteli apsauga

Recent studies have explored small-molecule inducers, selenium-based compounds, and targeted nanodelivery approaches to restore GPX4 function or mimic its antioxidant capacity in ferroptosis-prone diseases, with therapeutic implications for OA and beyond ( In summary, GPX4 acts not only as a terminal antioxidant enzyme but also as a hub that integrates transcriptional, post-transcriptional, and nutritional cues

This work was supported by GKLCDSM-20240101-02

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Forte A, Baldessarini RJ, Tondo L, Vzquez GH, Pompili M, Girardi P