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Because of its high prevalence in conjunction with obesity, diabetes, and insulin resistance, NAFLD has been increasingly appreciated as a hepatic manifestation of metabolic syndrome and is the leading cause of death from liver disease in the United States 1,2,3

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VEGFR2 activation can also stimulate angiogenesis - the formation of new blood vessels - which is essential for tissue repair and regeneration

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liver damage leads to reduced synthesis of coagulation factors, further exacerbating coagulation dysfunction [16]