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Its ability to penetrate the BBB, its metabolic stability, and its significant efficacy in restoring cognitive and motor functions in animal models of AD and PD, likely through modulating the HGF/c-Met system, position it as a highly promising therapeutic candidate for these debilitating neurodegenerative diseases [2]

This enhanced potency at the receptor level, combined with a peptide half-life superior to GHRP-6, makes GHRP-2 the preferred reference compound in research models requiring maximal pituitary GHS-R1a activation

Chiodini, G

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