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This data should be interpreted alongside biomarkers (glucose, insulin, lipids) and clinical history to build a comprehensive personalization picture
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J Rheumatol, 2007
Administered GLP-1 receptor agonists amplify residual incretin pathways in a glucose-dependent manner: they enhance insulin release when glucose is elevated, and taper as glucose normalizes, substantially lowering the risk of hypoglycemia compared with insulin secretagogues or intensive insulin regimens
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