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parkeri from ROS, we hypothesized that if the antimicrobial effects of BSO were mediated through ROS, bacterial abundance would be increased upon inhibiting nitric oxide (NO) production

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they recruit cAMP response element-binding protein (CREB)-binding protein (CBP) or receptor-associated coactivator 3 (RAC3), to facilitate the transactivation of NRF2 target genes [146, 147]

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In addition, the activation of GABA B receptors depresses the GABA A receptor-mediated Ca 2+ increase and therefore the GABA B pathway is likely supervising the entry of Ca 2+ [6]