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The estimates cited apply primarily to patients who are not taking drugs that affect hepatic metabolizing enzyme systems

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This suggests copper peptides may work through at least four mechanisms : DPC proliferation, anti-apoptosis, VEGF stimulation, and TGF-1 suppression

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This atrophy and loss of function are often a primary cause of neuropathology, as for example is caused by substantial decreases in expression and function of glutamate transporters in Wernicke-Korsakoff encephalopathy or toxic brain damage