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Furthermore, S-nitrosylation of specific targets can either promote (e.g., inhibiting GPX4) or inhibit (e.g., activating survival pathways) ferroptosis

10.1016/j.intimp.2024.112697 Summary Keywords experimental autoimmune myocarditis, levocarnitine, metabolic remodeling, mitochondrion, PI3K/AKT Citation Yang S, Li X, Lu Z, Xu X, Guo Z and He B (2026) Levocarnitine improves cardiac energy metabolic remodeling in myocarditis mice

Upon administration of glutamate and its antagonist (i.c.v.), glutamate strongly inhibited whereas APV promoted pulsatility among surface and penetrating arteries (Among surface arteries, Glutamate vs

Allergies pose another significant consideration