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GHK-Cu works through entirely different pathways (tissue repair, angiogenesis, anti-inflammation) than minoxidil (vasodilation) or finasteride (DHT suppression), so the three stack well
GHK-Cu exerts its effects primarily through gene expression modulation, influencing approximately 31% of the human genome by upregulating genes involved in antioxidant responses, DNA repair, protein metabolism, and cellular growth while downregulating inflammatory mediators, fibrinogen synthesis, and pathological processes including cancer metastasis
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