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Endogenous GLP-1 is secreted in small amounts from intestinal L-cells in response to nutrient stimuli and has a circulating half-life of less than two minutes due to rapid DPP-4mediated degradation

The combination of precise dosing and optimized nutrition produces outcomes that neither approach achieves alone

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Research is currently underway to see if taking oral weight-loss medication (such as bupropion-naltrexone) can help ease the transition off GLP-1 receptor agonists and reduce the amount of weight that's typically regained after stopping them

In contrast to previous generations of anti-obesity medicines, which had limited clinical efficacy, GLP-1-RAs engage the intricate neuroendocrine mechanisms that regulate body weight, resulting in safe and long-term weight loss