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Abstract Objectives: Hepatotoxicity is a severe outcome of methotrexate (MTX) therapy, limiting its clinical use and contributing to its related morbidity and mortality

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Interaction between nanoparticles generated by zinc chloride treatment and oxidative responses in rat liver

GSH adaptive response prevents CS induced GSH depletion There appears to be two distinct phases of the GSH adaptive response to CS exposures starting with an initial GSH adaptive response and then a maintained GSH response in the ELF

Therefore, therapeutic approaches that use nutrient addiction to target metabolic vulnerabilities may be effective in overcoming ferroptosis resistance