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It has been shown to counteract NSAID-induced ulcers, esophagitis, and inflammatory bowel conditions like colitis by promoting epithelial integrity, increasing mucosal blood flow, and regulating the gut-brain axis

The presence of these enzymes seems in itself to be good evidence that they are somehow required to optimise GSH-dependent reduction of DHA, although other functions cannot be ruled out

PubMed CrossRef Stewart R, Hirani V

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Neurosci Lett 402:137141 Droge W (1993) Cysteine and glutathione deficiency in AIDS patients: a rationale for the treatment with N -acetyl-cysteine