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Clathrin-mediated endocytosis (CME) usually requires arrestin to act as a scaffolding protein to link the receptor to the 2-adaptin subunit of adapter protein 2 (AP2), which then targets the receptor to clathrin-coated pits ( via clathrin-coated pits is an arrestin-independent process for GLP-1R ( The published data regarding agonist-induced internalization of GIPR are just as conflicting as, if not more so than, that for GLP-1R

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