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Helicobacter pylori infection: An overview of bacterial virulence factors and pathogenesis

Results BHBA Treatment Promotes BDNF Expression and Changes Histone H3K4 Trimethylation and Histone H2AK119 Mono-Ubiquitination in Bdnf Promoters Firstly, we verified the increase of BDNF levels in primary hippocampal neurons following treatment with 2 mM BHBA (Figure 1A)

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Based on available evidence, SCFAs exert protective effects by activating the Nrf2/GPX4 pathway to inhibit lipid peroxidation, while LPS acts through the dual mechanisms of suppressing the antioxidant defense system (GPX4) and disrupting iron metabolic homeostasis