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These changes persisted or were only partially reversible except for the changes caused by empagliflozin and sotagliflozin which showed full reversibility upon discontinuation

Resets the ubiquitin-proteasome system to clear damaged proteins

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FSP1 Is a Glutathione-independent Ferroptosis Suppressor

Furthermore, frataxin deficiency disrupts NRF2-mediated antioxidant responses, reducing the expression of key detoxifying enzymes such as superoxide dismutase 2, glutathione peroxidase, and catalase, thereby diminishing the cells capacity to counteract the elevated oxidative burden [126]