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These results suggest that iron chelators can mitigate neuronal damage by limiting iron-induced oxidative stress and ferroptosis ( 2+ levels and inhibit ferroptosis, current chelators lack tissue and cellular specificity and may interfere with neurophysiological functions, thereby limiting their clinical applicability
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Rodent wound biology differs materially from human wound biology, and a local implanted compartment is not a subcutaneous injection
They work on the biology of appetite rather than trying to override it through willpower alone
Further research into novel preventive and therapeutic strategies is urgently needed, especially for CMS and HAPH
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