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In dermal wound models, GHK-Cu treatment accelerates wound contraction, stimulates collagen synthesis (types I and III), and promotes the formation of organized granulation tissue

434,435,436 Glycogen synthesized from glycolysis-derived G6P also repolarizes macrophages to an inflammatory phenotype via the pentose phosphate pathway and UDPG-P2Y14 signaling pathway

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[75] reported that the traditional Chinese medicine Si-Ni-San reduces FASN expression by activating AMPK, which inhibits the activity of the transcriptional coactivator p300 and transcription factor SREBP-1c, thereby decreasing lipid accumulation and impairing lipid metabolism through a novel mechanism