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Treatment with inhibitor ferrostatin-1 (Fer-1) effectively suppresses high hydrostatic pressure-induced VSMC inflammation, as evidenced by reduced CXCL2 expression, and prevented endothelial dysfunction-associated phenotype switching marked by decreased AKR1C2 expression

It also introduces potential airborne contaminants

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Step 1 Remove the Source of Exposure: Recovery cant meaningfully begin while exposure continues

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