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Postnatal intracerebral administration of the GABA A receptor agonist clonazepam rescued deficient social novelty in mice that were exposed to VPA on E12.5, whereas the GABA B receptor agonist baclofen did not ( N -methyl-D-aspartate receptor antagonist MK-801 or metabotropic glutamate receptor 5 antagonist MPEP was similarly effective in restoring GABAergic signaling in rodents that were exposed to VPA on E12-13 (81, 86, 102)

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