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Though GLP-1 RAs have been recognized to improve glucose tolerance and induce sustained secretion of insulin after discontinuing therapy, yet no study has been conducted to determine whether Ex-4 plays a continuing role in protecting against stroke after cessation of therapy and the potential mechanisms

We hypothesised that the metabolite GLP-1(936) (previously believed to be biologically inactive) exerts a direct inhibitory effect on glucagon secretion and that this mechanism becomes impaired in diabetes

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