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Mechanistic studies have linked celastrols protective effects to pathways such as the reduction in HO-1-mediated autophagy and the SIRT1-mediated inhibition of the EZH2-related Wnt/-catenin pathway [22, 39]

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In PCOS animal models, free fatty acids in ovarian tissue increase, glucose oxidation is inhibited, free fatty acid oxidation is enhanced, and ROS increase through the TCA cycle, leading to IR (156)

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