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CONCLUSIONS GLP-1 RAs and multi-agonist therapies combining GLP-1 with GIP and/or GCG agonism embody transformative therapeutic approaches for MASLD/MASH, addressing multiple pathogenic pathways through complementary mechanisms of action

For typical once-daily prednisone, no timing changes are needed

The possibility of direct action on CSF dynamics by GLP-1 receptor activation is an area that warrants further exploration

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