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Experimental models show that GLP-1 receptor activation inhibits NF-B signaling, thereby reducing transcription of pro-inflammatory mediators including IL-1, IL-6, and TNF-

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A 2 L sample was used for protein concentration analysis with a BCA assay kit (Thermo Fisher Scientific, Waltham, MA)

Many ASD children present new types of mitochondria dysfunction, e.g., metabolic disturbances (inborn errors of amino acids metabolism in maple syrup disease (MSUD), dysregulation of the immunological system (ASD development may be connected with an increase in proinflammatory cytokines in natal-and perinatal periods) and/or inflammation in the nervous system, and oxidative stress (increased production of the reactive oxygen species)

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