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Both stacks exploit the same GHRH+GHRP synergy, but differ in the GHRH component: CJC-1295 + Ipamorelin : CJC-1295 (with DAC) has a prolonged half-life (~8 days) due to its binding to albumin

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Adenovirus-mediated overexpression of CD36 enhances hepatic fatty acid uptake and fat accumulation [22], while liver-specific knockout of CD36 decreases hepatic lipid levels in both genetic and diet-induced steatosis [23]