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[28] reported that in early Th17 differentiation, H3K4me1 is deposited at Th17-related gene regulatory regions by specific histone methyltransferases, priming enhancers for subsequent binding by RORt

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Keywords MC4R deficiency , genetic obesity , GLP-1 receptor agonists , leptin-melanocortin pathway , weight management 0 1 INTRODUCTION Genetic obesity is broadly divided into syndromic obesity and non-syndromic obesity, both of which can be caused by monogenic, polygenic, or chromosomal abnormalities [1]

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