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In addition to mitochondrial dysfunction, chelators that redox cycle (Dp44mT) [16] might exacerbate stress by induction of redox-active iron species that can damage cellular structures such as lysosomes [96]

Annexin V and PI staining was significantly increased in HSV-1-infected U373 cells compared to that in mock-infected cells, indicating that HSV-1 induced apoptotic cell death (Fig