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In summary, the crosstalk between autophagy and ferroptosis emerges as a key event in diverse liver diseases, including those associated with MS, NAFLD, NASH, ALI, diabetic liver damage and liver cell senescence

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Indirect Comparisons No indirect comparisons were identified or submitted by the sponsor

It activates the tyrosine kinase activity of IGF-1 receptor (IGF-1R), induces downstream MAPK/ERK and PI3K/Akt pathways, promotes the expression of cell cycle proteins (Cyclin D1, E), and accelerates the transition of cells from G1 phase to Sphase

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