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Glucoselowering by DPP4 inhibitors may therefore be mediated via portal vein GLP1R signalling and GLP1 sensing targeting neural pathways, and may explain the relatively prominent effects relative to the GLP1 plasma levels that DPP4 inhibitors induce in terms of reductions in plasma glucose concentrations compared with those achieved when employing GLP1RAs

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[27] In some cases, a bolus of plain IV solution (i.e