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This sophisticated crosstalk, where parallel pro-apoptotic pathways (ATF4-driven induction and CHOP-mediated fine-tuning) converge on the CHAC1 promoter, allows for precise control of cell fate under prolonged ER stress (Nomura et al., 2020)

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[DOI] [PubMed] [Google Scholar] 177.Polkinghorn W.R., Parker J.S., Lee M.X., Kass E.M., Spratt D.E., Iaquinta P.J., Arora V.K., Yen W.F., Cai L., Zheng D., et al

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