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In contrast, the key lipogenic and fibrogenic genes that are suppressed by Chac1 overexpressioninduced hepatic GSH depletion, including Srebf1 , Scd , Dgat2 , Spp1 , Col1a1 , and Sox9 , were upregulated during disease progression (Figure 6, E and F)
In the current clinical context, the benefitrisk profile in patients at high cardiometabolic and cardiorenal risk remains clearly favorable
16 Administration of GSH resulted in a significantly higher urine volume (approximately 20%) on the first and second postoperative days, with a trend to lower blood urea nitrogen and plasma creatinine concentrations in the GSH group that did not reach statistical significance
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