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10.1089/ars.2011.4328 80 CudkowiczM

Agnieszka Adamska, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

And as human beings we generally love familiarity and resist change

This therapy involves maintaining specific levels of blood glucose, lactate, and -hydroxybutyrateachievable through diet and exercisecoupled with redox state maintenance via NAD supplementation, modulation of the cytosol-mitochondria reducing equivalent shuttle (related to NADH, i.e., DEN therapy), and Na + /K + -ATPase activation

GSTA1 and GSTM1 can also form protein complexes with JNK in a mechanism similar to GSTP1, but their inhibitory activity against JNK is rather weak [2,36]