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Immunohistochemistry analysis revealed that Sorafenib treatment significantly reduced the levels of SLC7A11 and Ki67, a proliferation marker, and increased the level of 4-HNE, a marker of ferroptosis, in tumor tissues (Fig

In laboratory settings, BPC-157 is investigated for its role in cellular signaling processes, including pathways associated with fibroblastic activity and epithelial system models

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Notably, this effect is epithelium-independent and involves the activation of the cAMP-PKA signaling pathway, which mediates ASM relaxation and counteracts BHR [11]