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In addition, there may be potential that these fragments are binding to these same receptors to elicit a zonulin-like effect in tight junction dysregulation, versus an inhibition of zonulin activity, although mechanistic studies need to be conducted to fully explore this possibility

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FOXO4-DRIs ability to remove these cells has shown potential in models of Alzheimers disease and other neurodegenerative conditions (5, 11)

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