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Moreover, in animal models, ALC has been shown to attenuate the release of pro-inflammatory mediators (IL-1, TNF) from activated microglia, to modulate neuroinflammation and to reduce the concentration of oxidative stress markers, increasing the concentration of glutathione (GSH), a powerful mediator with antioxidant proprieties [27,28]

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At the same time, concerns persist regarding the gastrointestinal side-effects commonly associated with GLP-1RAs, which may limit their applicability in patients with coexisting IBD

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This in itself has implications for the way that it is classified, prescribed and administered