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The outcome suggested that a transient and limited increase in gut permeability compared with the unregulated breach of the gut barrier function due to the TJ deregulation and epithelial cell apoptosis can rather lead to a dominant regulatory immune response to gut microbiota antigens and to a net anti-inflammatory effect

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Of course, this should be taken with a grain of salt

Furthermore we examined the mRNA expression level of fat related genes including sterol regulatory element-binding factor 1 (Srebf1), Acetyl-CoA carboxylase (Acc1), and long-chain acyl-CoA dehydrogenase (Lcad)