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The BPC-157 peptide is documented in peer-reviewed literature as acting through at least two distinct, non-redundant molecular pathways simultaneously which may explain its unusually broad tissue-repair activity profile

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Interfering with this interaction, therefore, allows p53 to cause senescent cells to enter apoptosis

[4] The presence of the hydroxyl functional groups and the hydrophobic hydrocarbons render the molecule amphiphilic

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