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We discuss how ME/CFS emerges from an intricate web of genetic vulnerabilities and environmental triggers, notably viral infections, leading to a complex series of pathological responses including immune dysregulation, chronic inflammation, gut dysbiosis, and metabolic disturbances

Interestingly, the expression of integrin 1 (CD29), TGF-1, and type I collagen is considerably lower in the fibroblasts of SUI patients than in those of donors without SUI (Li et al