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Among its components, STAT3 plays a central role by upregulating matrix metalloproteinases (e.g., MMP-2 and MMP-9), stimulating the proliferation of fibroblast-like synoviocytes, and suppressing apoptotic pathways (Ciobanu et al

DPP4 degrades endogenous GLP1 within minutes, and DPP4 inhibitors are approved for treatment of Type 2 diabetes (Deacon, 2020) as an indirect way to stimulate GLP1 receptors (upina et al., 2020)

No difference in maximal respiratory capacity was observed with either TNF- or NR co-treatment (Fig 2F)

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