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Secondary endpoints were univariately compared between the GLP-1-RA and UCWM groups

This phenomenon could be attributed to the fact that ethanol exposure accelerated the pathological process of AD, which in turn damaged the antioxidant capacity in APP/PS1 mice

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For instance, LePHGPX in transgenic tobacco plants protected against DNA fragmentation, membrane integrity loss, and oxidative stress induced by necrotrophic fungi, mirroring anti-apoptotic roles observed in animals (Chen et al., 2004)