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Apart from the low propensity of the GSAAT(C168S) and GSAAT(C190S) mutants to dimerize, these single substitution mutants not only failed to promote redox-dependent dimerization and oxidation of monomeric GSAAT, but even prevented it

Therapeutic Research 56 (7), 656670

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The NAPstar3b variant was generated by site-directed mutagenesis on the pDONR207 NAPstar3 plasmid using the primer pair Pr1/Pr2 (Supplementary Table 6)

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