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In fact, impaired GSH synthesis or reduced GSH import into the periplasm is connected to several phenotypes, similar to those found in cells lacking the DsbA/DsbB- or DsbC-system (Fabianek et al., 2000

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Extracellular GS-Es are degraded sequentially by cell surface enzymes, -glutamyl transpeptidase (GGT) and dipeptidases, to cysteinyl-electrophile conjugates that are N-acetylated to mercapturic acid in the kidneys

Complications of High Blood Protein If high blood protein is left untreated or is not managed effectively, it can lead to complications, including: Kidney Damage: Persistently high protein levels can strain the kidneys, potentially leading to kidney disease or failure

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