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Consequently, CHAC1 serves as a central mediator connecting ISR (ATF4) to the antioxidant response (NRF2) and metabolic adaptation (SLC7A11), thereby enabling cells to dynamically fine-tune redox homeostasis and maintain metabolic flexibility during stress (Kre et al., 2023)
Autologous CD19R(EQ)28/EGFRt+ Tn/mem cell products were administered by intravenous infusion in a single dose following a 3-day lymphodepleting regimen
L., Calanna, S., et al
A confocal microscopic study of solitary pulmonary neuroendocrine cells in human airway epithelium
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